(732) 345-1377 info@monmouthpain.com

When a patient has degrees of cellular and or vascular damage, the soft tissue site can be in a potentially anaerobic state.

Cells deprived of oxygen or blood supply have a potential increase lactic acidosis and therefore amplify local cellular damage. Normal physiologic healing requires growth factors and cytokines to be released at the wound site.

These cell mediators called inflammatory cells to the wound which clean up damaged tissues, fight bacteria and stimulate fibroblasts and vascular cells to try and heal the zone from injury.

The physiologic concept is that Low Level Laser Therapy improves cellular metabolism and accelerates the process of debris cleaning, improves neutrophil bacterial clearing and hastens cellular division.

All stages of wound healing, hemostasis, inflammation, cell proliferation and migration, collagen synthesis, wound contraction and wound remodeling proceed more rapidly and more effectively. There are biological processes that take place in tissues that have been shown to respond to Low Level Laser Therapy in the 630-640 nanometer range.

Cold Laser Therapy with Erchonia PL 5000 (PL-5) is medically necessary in healing patients with pain, inflammation and tissue healing. This form of rehabilitation is used on the patient in conjunction with a conservative treatment protocol. For more information on Cold Laser Therapy, also known as Low Level Laser Thearapy complete the form above or contact our office at 732-345-1377.


1 -Simunovic Z, “Low energy laser therapy with trigger Points technique” J Clin Laser Med Surg 1996 Aug;14(4);163-7
2- Simunovic Z, “Wound Healing of animal and human body sport and traffic accident injuries using low-level laser therapy treatment: a randomized clinical study of seventy four patients with control group” Clin Laser Med Surg 2000 Apr; 18(2):67-73
3- Stelian J, “Improvement of pain and disability in elderly paitents with degenerative osteoarthritis of the knee treated with narrow-band light therapy,” J Am Geriatr Soc !992 Jan; 40(1):23-6